February 28, 2024

Study shows sinusitis may raise risk of rheumatic disease by 40%

People suffering from the common inflammatory condition sinusitis may be at 40 per cent risk of developing a subsequent diagnosis of rheumatic disease, particularly in the five to 10 years preceding the start of symptoms, finds a research.Sinusitis refers to inflammation of the lining of the sinuses, the small, air-filled cavities behind the cheekbones and forehead.

Researchers from the Mayo Clinic and the Harvard Medical School in the US showed that a history of sinusitis was associated with a 40 per cent heightened risk of any new diagnosis of rheumatic disease.

The strongest association was seen for systemic autoimmune rheumatic diseases, such as antiphospholipid syndrome -- seven-fold increased risk, and Sjogren’s syndrome -- more than double the risk.

Antiphospholipid syndrome is a blood clotting disorder, while Sjogren's is a condition that affects the body's production of fluids, such as spit and tears.

The findings, published in the open access journal RMD Open, also showed that acute sinusitis was associated with an 18 per cent heightened risk of seronegative rheumatoid arthritis (symptoms but no detectable antibodies).

"Overall, these findings point towards a role for sinus inflammation in the presentation, and possibly pathogenesis, of rheumatic disease," the researchers said.

"Bacterial pathogens, such as those involved in sinusitis, might have a role in rheumatic disease, added to which sinusitis is associated with speeding up artery hardening, lending extra weight to its potential inflammatory effects," they explained.

The study sample included 1,729 adults, newly diagnosed with a systemic autoimmune rheumatic disease, such as rheumatoid arthritis, antiphospholipid syndrome, and Sjogren's syndrome; or vasculitis (blood vessel inflammation), such as giant cell arteritis (temporal artery inflammation) and polymyalgia rheumatica (muscle pain and stiffness).

Each of these patients (average age 63; two thirds women) was matched with 3 people (5187 in total) with no rheumatic disease, based on age at diagnosis and sex.

The results showed the risk was 70 per cent higher in the 5-10 years preceding symptom onset. 

Frequency of sinusitis episodes also increased the risk. For example, those experiencing 7 or more were nearly 5 times as likely to be diagnosed with systemic autoimmune disease, nearly 9 times as likely to be diagnosed with Sjogren’s syndrome, and twice as likely to be diagnosed with vasculitis.

The team, however, noted that "this is an observational study, and therefore no definitive conclusions can be drawn about causal factors. And reverse causation, whereby the rheumatic diseases themselves increase the risk of sinusitis, can't be ruled out".

https://in.investing.com/news/study-shows-sinusitis-may-raise-risk-of-rheumatic-disease-by-40-4039944

Tata Institute comes up with a ₹100 drug to prevent cancer recurrence; doctors to start human trials soon

Tata Institute has come with drugs to stop cancer recurrence

Researchers at the Tata Institute in Mumbai announced the development of a novel drug aimed at preventing cancer recurrence, according to a report by Hindustan Times. The researchers and doctors at the institute have reportedly worked for 10 years and have now come up with a tablet which they claimed would stop the occurrence of cancer for the second time in patients.

 

The tablet is priced at 100 per piece, according to Business Standard.

 “We did a little experiment in which we took human breast cancer cells and implanted them in immunodeficient mice," said Dr Indraneel Mittra, the onco-surgeon-turned-scientist who led the TMH doctors in the study, as quoted by Hindustan Times. “Within six weeks, a small tumour was formed. We divided the mice into three categories according to the cancer treatment—chemotherapy, radiotherapy and surgery. We found that all the three treatments increased chromatin in the mouse brain."

 

“We found that a combination of resveratrol and copper helped in destroying chromatin. We used the combination to be given orally in our studies and found that it prevented metastasis," said Dr Mittra, who added that while this was the outcome of the experiment on mice, the team would now do human trials, as per the HT report. Dr Mittra joined TMH in 1982 as the country’s first “surgeon scientist".

 

Mittra further explained that after confirming the presence of chromatin particles following cancer treatment, the team’s focus shifted towards reducing treatment-related toxicity (side effects) in various human cancers, such as stomach, brain, oral, and blood cancers through clinical trials, as per the Hindustan Times report.


https://www.livemint.com/science/health/tata-institute-comes-up-with-a-rs-100-drug-to-prevent-cancer-recurrence-doctors-to-start-human-trials-soon-11709098302960.html

IIT Madras develops 1st India-specific AI model to determine foetus age

Accurate age of a foetus -- gestational age -- helps in providing proper care of pregnant women and also to determine precise delivery dates

In a first, researchers at the Indian Institute of Technology (IIT) Madras have developed an Artificial Intelligence (AI) model that is specific to Indian population and can accurately determine the age of a foetus in a pregnant woman, in her second and third trimesters.

Currently, physicians in India determine the gestational age using a formula developed for Western population, increasing chances of error.

The new model called ‘Garbhini-GA2' accurately estimates the age of a foetus for the Indian population, reducing error by almost three times, said the team of researchers, including from the Translational Health Science and Technology Institute (THSTI), Faridabad on Monday.

It is “the first late-trimester gestational age estimation model to be developed and validated using Indian population data”, they noted in the study, published in the peer-reviewed journal Lancet Regional Health Southeast Asia.

The new model can also improve the care delivered by obstetricians and neonatologists, thus reducing maternal and infant mortality rates in India.

The research is also part of ‘Interdisciplinary Group for Advanced Research on Birth Outcomes -- DBT India Initiative' (GARBH-Ini) programme.

“GARBH-Ini is a flagship programme of DBT, and the development of these population-specific models for estimating gestational age is a commendable outcome. These models are being validated across the country,” said Dr. Rajesh Gokhale, Secretary, Department of Biotechnology (DBT), Government of India, in a statement on Monday.

“IIT Madras has been contributing towards solving healthcare problems at the grassroots and local level with the aim of enhancing public health in India. To this end, we are utilising advanced data science and AI/ML techniques to build tools to predict unfavourable birth outcomes. The first step towards this is to develop accurate GA models that perform significantly better than currently used models designed using Western populations,” added Dr Himanshu Sinha, a Coordinator at the Center for Integrative Biology and Systems Medicine, IIT Madras, and who led the data science work for this research.

Garbhini-GA2 used three routinely measured foetal ultrasound parameters, and was developed using GARBH-Ini cohort data documented at Gurugram Civil Hospital, Haryana, and was validated in an independent cohort in South India.

Once validated in prospective pan-India cohorts, this Garbhini-GA2 can be deployed in clinics across India, improving the care delivered by obstetricians and neonatologists, thus reducing maternal and infant mortality rates in India, the researchers said.

“Improving the gestational age accuracy is a critical component of the broader goals of the GARBH-Ini study, which aims to reduce the adverse pregnancy outcomes,”said Dr Shinjini Bhatnagar, Principal Investigator of the GARBH-Ini programme and a Distinguished Professor, at THSTI.

https://www.tribuneindia.com/news/health/iit-madras-develops-1st-india-specific-ai-model-to-determine-foetus-age-594763


Common anesthetic in low doses can improve symptoms of depression, find researchers

Low doses of ketamine can restore social deficits by restoring function in anterior insular cortex, says study.

Well-being is important for everyone, especially when we are lonely or alone. Depression is a serious problem for many people, and finding an effective therapy is crucial.

A study published in Molecular Psychiatry found that low doses of ketamine, a common anaesthetic, can restore social deficits by restoring function in the anterior insular cortex.

Ketamine is often used at low doses to treat depression, but its actions in the brain remain relatively unclear. Generally, ketamine refers to a mix of two different forms of ketamine: (S)-ketamine and (R)-ketamine.

These two molecules are mirror isomers or enantiomers--they have the same molecular formula, but their three-dimensional forms are mirror images of one another.

Although they usually occur as (S) and (R) pairs, they can also be separated into either (S)-ketamine or (R)-ketamine. Each is beneficial in treating depression, although their specific effects vary.

When the research team decided to test the effects of (S)-ketamine and (R)-ketamine on depression-like symptoms in mice, they first had to decide on an appropriate model. Given that depression and social impairments can be induced by long-term social isolation, they chose a chronic (at least 6 weeks) social isolation mouse model.

The researchers then used a method that allowed them to directly compare neuronal activation throughout the entire brains of mice treated with (S)-ketamine, (R)-ketamine, or saline (as a control) directly after behavioural tests.

"In this way, we were able to observe differences between (S)-ketamine and (R)-ketamine treatments in terms of neuronal activation across the whole brain, without having a predefined hypothesis," said lead author of the study Rei Yokoyama.

"Notably, we found that chronic social isolation led to decreased neuronal activation in the anterior insular cortex--a brain region that is important for emotional regulation--during social contact, and that (R)-ketamine, but not (S)-ketamine, reversed this effect." The researchers also found that mice treated with (R)-ketamine were better at recognizing unfamiliar versus familiar mice in a social memory test, indicating improved social cognition. Moreover, when neuronal activity was suppressed in the anterior insular cortex, the (R)-ketamine-induced improvements disappeared.

"These findings highlight the importance of the anterior insular cortex for the positive effects of (R)-ketamine on social impairments, at least in mice," said Hitoshi Hashimoto, senior author of the study.

"Together, our results indicate that (R)-ketamine may be better than (S)-ketamine for improving social cognition, and they suggest that this effect is dependent on restoring neuronal activation in the anterior insular cortex." 

https://www.tribuneindia.com/news/health/common-anesthetic-in-low-doses-can-improve-symptoms-of-depression-find-researchers-594479

Why bariatric surgery is effective for type 2 diabetes management


  • Researchers report that bariatric weight-loss surgery provides more long-term benefits for managing type 2 diabetes than lifestyle changes.
  • They said that bariatric surgery also improves cholesterol and triglyceride levels more effectively than medical or lifestyle modifications.
  • Currently, people with body mass index readings of under 35 do not qualify for the surgery under most health insurance company guidelines.

Bariatric surgery provides more benefits than lifestyle changes in managing type 2 diabetes, according to a study completed at the University of Pittsburgh School of Medicine and published today in the journal JAMA.

As part of the research, participants with type 2 diabetes and obesity enrolled in one of four randomized clinical trials completed between May 2007 and August 2013.

The participants underwent bariatric surgery or completed a medical and lifestyle program based on established interventions known to reduce diabetes risk.

Interventions included physical activity, nutrition tracking, enhanced engagement with a healthcare team, stress management, support groups, and medication. The trials occurred before the availability of GLP-1 receptor agonist medications such as Ozempic for diabetes management and weight loss.

The researchers followed most of the participants for 12 years.

Findings in the weight loss surgery and diabetes management study

The researchers reported that bariatric surgery improved cholesterol and triglyceride levels more effectively than medical or lifestyle modifications. High cholesterol levels are a risk factor for heart disease.

Participants who had the surgery also consistently had lower HbA1c levels, indicating better blood sugar control, at every follow-up point, despite starting the study with higher baseline levels.

The findings also included:

  • At the seven-year follow-up, 18% of participants achieved diabetes remission, compared to 6% in the medical and lifestyle groups.
  • At the 12-year follow-up, participants who had surgery achieved an average of 19% weight loss, compared to slightly less than 11% in the medical and lifestyle intervention group.
  • Anemiafractures, and adverse gastrointestinal symptoms, such as nausea and abdominal pain, were more common in those who received bariatric surgery.

Participants who did not achieve diabetic remission still had better blood sugar control and used less diabetes medication than those who underwent lifestyle changes, the researchers noted.

Reaction to the bariatric surgery study

“In my practice, I have seen patients who have weight loss surgery be able to discontinue their diabetes, high blood pressure, and lipid-lowering medications,” said Dr. Mir Ali, a bariatric surgeon and medical director of MemorialCare Surgical Weight Loss Center at Orange Coast Medical Center in California who was not involved in the study.

The confirmation from the study results that bariatric surgery is more effective than lifestyle interventions was not surprising to Dr. Anita Courcoulas, the chief of the Section of Minimally Invasive Bariatric and General Surgery at the University of Pittsburgh School of Medicine and one of the authors of the study.

What is new/novel is that this is the largest study of its type and the length of follow up of 7 to 12 years is the longest,” she told Medical News Today. “As well, this study includes 37 percent of participants with BMI (body mass index) less than 35 with class 1 obesity and also finds long-term superiority of surgery over medical/lifestyle treatment in this subset. These findings combined with existing evidence lend very strong support for the use of bariatric surgery to treat type 2 diabetes in people who cannot achieve glycemic control by non-surgical means.”

Dr. Eliud Sifonte, an endocrinologist at NYU Langone Medical Associates — West Palm Beach and Delray Beach in Florida who was not involved in the study, agreed.

“This study confirms previously known findings showing the benefits of bariatric surgery in those with metabolic diseases like diabetes and the efficacy of early interventions in the progress of a diabetic,” he told Medical News Today. “It also confirms the weight-centered approach to treatment of metabolic derangements as compared to the traditional glucose-centric approach.”

“I believe this confirmation could be key to helping some patients make the decision to undergo bariatric surgery particularly since it presents the possibility of remission, which is something many people with new onset diabetes are interested in,” Sifonte added. “In my practice I typically discuss bariatric surgery with those patients who have a history of obesity with metabolic complications regardless of the typical threshold.”

BMI and bariatric surgery

The researchers reported that the study’s results were consistent across weight class groups, indicating that surgery was equally beneficial for people with body mass indexes (BMI) below and above 35 kg/m2.

Typically, physicians do not recommend surgery for people below the 35 BMI level.

“BMI is not an ideal marker of metabolic disease,” said Dr. Mitchell Roslin, the chief of bariatric surgery at Northwell Lenox Hill Hospital in New York who was not involved in the study. “In fact, even high sugar misses a lot of people. All the big killers in the West are linked to metabolic disease. This includes heart disease, cancer, and neurological degenerative diseases.”

“Bariatric surgery that provides a long-lasting control mechanism that better matches the brain and GI tract, reduced hunger and prolonging satiety, is the best and perhaps only treatment to provide long-term control of metabolic syndrome,” Roslin told Medical News Today. “While obesity makes metabolic [disease] more common, there are many with smaller BMI who have metabolic disease. Additionally, as people age, metabolic disease increases.”

“Thus, [I have] no reluctance to offer bariatric surgery to low BMI patients with metabolic syndrome,” he added. “Like any therapy, [some] things will be prevented and others, more likely. The advantage is a lower risk of heart disease and cancer and [but] an increased risk of osteoporosis and anemia. In my mind, this risk can be mitigated with follow-up after surgery, diet, and exercise.”

The researchers did not find differences in mortality or major cardiovascular events in different weight class groups.

Insurance payments for weight loss surgery

In many cases, the final word on having bariatric surgery is not with the doctor or the patient.

“The decision is up to their health insurance company,” Mir told Medical News Today. “Many times, the insurance companies are quite strict with approvals. People with a BMI of under 35 often do not get approved. Sometimes, if there are other health conditions, such as diabetes, we can ask them to approve payment. More often, they deny the claim.”

Ali said he has been working with other physicians to try to get the insurance companies to lower the BMI threshold for weight loss surgery, but so far has not had any luck.

https://www.medicalnewstoday.com/articles/why-bariatric-surgery-is-effective-for-type-2-diabetes-management#Insurance-payments-for-weight-loss-surgery

How does psoriasis spread throughout the body? Study helps explain


  • A new study investigates gene expression in the skin tissue of people with psoriasis.
  • The researchers used a relatively new technique called spatial transcriptomics.
  • This approach helps map differences in gene expression in healthy and affected skin.
  • The researchers said they identified immune cell changes between healthy and diseased skin.

Psoriasis is one of the most common immune-mediated inflammatory diseases, affecting up to 3% of people worldwide.

Aside from its familiar dermatological symptoms, psoriasis also affects other organs and systems.

People with the condition have an increased risk of metabolic syndromemental health conditions, and cardiovascular problems.

In addition, about 1 in 3 people with psoriasis develop psoriatic arthritis.

How psoriasis spreads throughout the body

While scientists have made headway in understanding and treating psoriasis, it is not yet clear why there are variations in symptoms between individuals.

Some people with psoriasis have plaques on just a small part of the body while others experience them over much larger sections of skin. Also, these plaques can vary substantially in thickness, scaliness, and redness.

Evidence shows that the risk of developing cardiovascular disease and psoriatic arthritis increases with skin symptom severity.

Therefore, it is vital to explore the mechanisms involved in symptom severity and how localized skin inflammation leads to systemic disease.

A recent study in the journal Science Immunology helps fill in some missing pieces by employing spatial transcriptomics.

“Our initial goal was to find measurable molecular signals that could tell us who is more likely to develop severe psoriasis as well as who is at higher risk of developing related disorders that often accompany psoriasis, such as arthritis and cardiovascular disease,” Dr. Jose Scher, a study co-senior investigator and a specialist in psoriatic disease at NYU Langone Health in New York, said in a press statement.

What is spatial transcriptomics?

Spatial transcriptomics is a relatively new technology that allows scientists to map cellular activity within human tissues precisely.

Standard techniques to measure gene expression often involve homogenizing tissue samples. This means that spatial information is lost.

With spatial transcriptomics, however, scientists can analyze gene expression in intact tissue. This provides detailed information about how gene expression varies in different regions of the sample.

In the current study, this technique allowed researchers to assess immune activity in different regions and layers of skin.

Results from the study on psoriasis spread

In the recent study, the scientists collected tissue biopsies from the flank, forearm, or buttocks of three healthy participants as well as 11 participants with psoriasis.

In total, the researchers analyzed 25 biopsies.

The researchers reported that in more severe cases of psoriasis certain cells were in different positions within the skin’s layers.

Immune cells in samples from healthy participants were particularly prevalent around follicles and blood vessels. However, these cells were closer to the skin’s surface in samples from people with psoriasis.

In particular, fibroblasts — a key source of inflammatory cytokines — and macrophages were more common in the upper layers of skin in severe disease.

In moderate-to-severe cases of psoriasis, the researchers noted increased gene expression in molecular pathways associated with the control of lipid levels and metabolism.

These pathways play a role in various metabolic dysfunctions, including type 1 diabetes, nonalcoholic fatty liver disease, cholesterol metabolism, and lipolysis in adipocytes.

The researchers identified this uptick in expression in both psoriatic lesion tissue and unaffected tissue from participants with psoriasis. This underlines one of the challenges in studying psoriasis — even if drugs successfully treat the more obvious external physical symptoms, molecular mechanisms may still be working against overall health.

These metabolic pathways, the authors suggest, may play some part in the increased risk of diseases such as type 2 diabetes. However, researchers will need to carry out more work to understand how this plays out.

The skin microbiome and psoriasis

Follicles are rich with commensal bacteria. They also act as a gateway for the entry of allergens and pathogens. Follicular regions also richly express cytokines and immune activation factors.

As the authors explain, animal studies show that bacteria living in follicles are important for immune surveillance. As an immune-mediated disease, these bacteria could provide another piece of the psoriasis puzzle.

This study demonstrated how immune cells are situated more distantly from follicles in psoriatic samples, which could interrupt the healthy interaction between commensals and the immune system.

Researchers said understanding which microbes reside in follicular regions within healthy and diseased skin might provide more insight.

Future study on the spread of psoriasis

Researchers said this study adds an important new tool for dermatology research because spatial transcriptomics effectively defined biologically relevant, distinct cellular “neighborhoods” in healthy skin.

“Our study serves as a valuable resource for the scientific community, offering the most comprehensive archive of cellular and molecular features involved in both diseased and healthy skin,” said Shruti Naik, PhD, a study senior co-investigator and an assistant professor in the departments of pathology and medicine as well as the Ronald O. Perelman Department of Dermatology at NYU Langone, in a press statement.

Medical News Today spoke with Axel Svedbom, PhD, a researcher at the Karolinska Institutet in Sweden who was not involved in the study.

One of the challenges of studying psoriasis, he explained, is that the condition “is quite heterogenous and it is possible that grouping all patients with psoriasis together masks important mechanisms that would be found if patients were more carefully phenotyped.”

He added that techniques, such as spatial transcriptomics, might help scientists move toward this goal and build more specific treatment plans for individuals.

“I think the next step is to move toward precision medicine. Combining data from clinicians, patients, and the lab will help us to identify the right treatment for the right patient and at the right time,” Svedbom said.

As technology advances and spatial transcriptomics gains higher resolution, the study authors said they hope to gather increasingly detailed information about skin in healthy and diseased states.

“Collectively, our findings underscore the value of spatially-resolved gene profiling in understanding emergent cellular ecosystems underlying health and disease,” they wrote.

Sunlight exposure linked to improved fertility outcomes, study finds

  • Results from a recent study suggest that moderate exposure to solar radiation during autumn and spring may help improve ovarian reserve in women ages 30–40.
  • It’s too soon to determine the effects of solar radiation on fertility outcomes, particularly in younger age groups.
  • Research is ongoing about which factors affect female fertility and how women can modify these factors.

Many people face fertility challenges, particularly those who are over 35.

Researchers are exploring various potential causes of infertility, such as environmental factors, and whether modifying these factors could have any affect on fertility outcomes.

A recent study published in Steroids examined how exposure to solar radiation influences female fertility. Researchers examined levels of a specific hormone called anti-Müllerian hormone (AMH) and how levels of this hormone varied during different times of the year.

The results were significant for women over 30 who experienced higher levels of AMH in the spring and autumn when there were moderate levels of solar radiation intensity.

However, researchers did not observe this effect in women under 30, indicating that solar radiation may affect female fertility more with age. More research is required to understand these factors and the potential clinical implications.

Sun exposure boosts female fertility

Researchers looked at how sun exposure influenced fertility among women of younger and older maternal age. This study was conducted in Israel and included 2,235 women mainly between 20 and 40 years old.

Researchers wanted to better understand the relationship between solar radiation exposure and anti-Müllerian hormone (AMH), an indicator of female fertility.

“When evaluating the fertility status of a patient, often, an anti mullerian hormone level is obtained. This level correlates to ovarian reserve. Studies have revealed that AMH declines with age, and therefore also does fertility,” non-study author Dr. Kelli V. Burroughs, a national media women’s health medical expert and department chair of OB-GYN at Memorial Hermann Sugar Land in Texas, explained to Medical News Today.

However, researchers of the current study note that AMH doesn’t necessarily reflect the quality of oocytes.

The study authors looked at participant data for 4 years. Researchers measured solar radiation in the central district of Israel using data from the Israeli Meteorological Service website.

Researchers found that AMH levels declined with age, so they divided participants into two groups: aged 20-29 years and 30-40 years. For women in the 20–29-year category, researchers did not find an association between AMH levels and the seasons or solar radiation intensity.

However, the results differed for women in the 30–40 year group. Researchers found that AMH levels increased for these women in the spring and autumn when there were moderate solar radiation intensity levels compared to the winter months when there were low solar radiation intensity levels.

The levels of AMH during months of moderate solar radiation exposure were overall higher than months that had high or low-intensity solar radiation levels.

They also found that participants in the 30–40-year group who had AMH levels collected during the summer months had much higher AMH levels than participants who had AMH levels collected during the winter months.

Researchers further divided participants into 30–35-year and 36-40-year groups. In the 30–35-year group, they did not find a significant correlation between solar radiation intensity or season and AMH levels. In the 36–40 group, they discovered that AMH levels were higher in the months of moderate solar intensity and higher in the summer compared to winter.

The results indicate that exposure to moderate solar radiation may be helpful for women in their 30s who are trying to get pregnant.

Dr. Burroughs noted the following:

“This study is interesting because it suggests for women between ages 30-40 there is a possible seasonal influence on the AMH driven by the amount of sunlight or UV exposure. The mechanism behind the correlation of AMH and seasonal UV light exposure is unknown, but the study revealed higher levels of AMH with moderate UV exposure during spring and fall. It was also noted that low and high levels of UV exposure had the opposite effect on AMH levels.”

Factors that influence female fertility

Fertility is complex and affected by many components. Sometimes, it’s possible to modify certain factors that may contribute to infertility.

For example, both obesity and being underweight can increase the risk of infertility. Smoking or heavy drinking can decrease fertility. Certain health conditions can also impact female fertility, such as:

Non-study author Dr. Kecia Gaither, MPH, double board-certified OB-GYN and maternal-fetal medicine specialist and director of Perinatal Services/Maternal Fetal Medicine at NYC Health in New York, told MNT:

“There are many factors which impact fertility — drugs, stress, female factors (endometriosis, fibroids, polyps, hormonal imbalances, PCOS etc) [and] male factors (i.e., low sperm counts). It’s important to note the environmental aspect — (i.e., plain sunlight exposure) as a factor correlating with positive reproductive health [or] outcomes.”

One non-modifiable factor that affects female fertility is advanced maternal age as the chances of successful conception begin to decline after 35.

Dr. Burroughs noted the following:

“More women than ever are delaying childbirth until their 30s and 40s for a variety of reasons including education, employment obligations, career-goals and increase[d] access to contraception. The result of delayed childbearing until the 3rd or 4th decade in life can impact fertility, because as a woman ages the ovarian reserve or number of eggs starts to decline. After the age of 35, a woman is considered advanced maternal age (AMA). This terminology reflects the correlation between age and declining fertility status.”

More research on the effects of sunlight on fertility needed

This study does have limitations. First, it doesn’t establish any causal relationship between the observed components.

Researchers also acknowledge that the lack of any significant association between AMH and seasons and participants between 20–29 years could be because of the difference in sample size between this group and the older group.

Researchers also acknowledge that they did not analyze luteinizing hormone (LH) and follicular stimulating hormone (FSH) hormone levels in women aged 26–30, and this could have impacted their findings in this area.

Researchers also note the possibility of confounding related to things like skin tone and cultural distinctions. Other factors like lifestyle and personal choices of participants could have also impacted the results.

The research also focused on one area of the world, so the results could be different if conducted in other countries. Researchers did not take into account the origin of participants in their analysis. Finally, researchers did not have access to certain clinical information, like reproductive history.

As research continues, study authors note that the potential benefits should be balanced with the possible risks of sun exposure, such as cancer or skin damage.

https://www.medicalnewstoday.com/articles/sun-exposure-may-improve-female-fertility#More-research-on-the-effects-of-sunlight-on-fertility-needed

Mobocertinib withdrawal: What it means for people with EGFR+ lung cancer


  • EGFR+ lung cancer is a type of lung cancer, not linked to smoking, that is caused by one of a number of nonhereditary gene mutations.
  • One mutation, exon 20, had no effective treatments beyond chemotherapy until a tyrosine kinase inhibitor called Mobocertinib (marketed as Exkivity) was licensed in 2021.
  • However, the manufacturers withdrew it from use in the United States late last year, and health authorities in the United Kingdom plan to follow suit next month.
  • EGFR+ campaigners are concerned that people in the United Kingdom with this mutation will now have no treatment options to extend life after chemotherapy.

Even though, according to the Centers of Disease Control and Prevention (CDC), 80–90%Trusted Source of lung cancer cases are linked to smoking, a significant number of lung cancers are not smoking-related.

Most of these are non-small cell lung cancers (NSCLC), caused by mutations of the epidermal growth factor receptorTrusted Source, termed EGFR+ lung cancers.

These cancers often present in younger people and have atypical symptoms, such as shoulder pain, or other musculoskeletal symptoms, rather than the coughing, breathlessness and recurrent chest infections that are usually seen in smoking-related lung cancers.

Several different nonhereditary mutations can lead to EGFR+ lung cancer. The most common are EGFR 19 deletion — where part of the gene is missing — and EGFR L858R point mutation, in which one nucleotideTrusted Source (small unit of DNATrusted Source) is altered.

Why mobocertinib is important for people with lung cancer

The exon 20 insertion mutationTrusted Source is the third most common cause of EGFR+ lung cancer, being responsible for up to 10% of cases. People with this mutation generally have a poorer prognosis than people with different mutations.

Apart from platinum-based chemotherapyTrusted Source, there were no effective treatments for exon 20 available in the United Kingdom, as the tyrosine kinase inhibitorsTrusted Source (TKIs) used to treat other EGFR+ mutations do not work against exon 20.

However, in 2021, a new TKI, mobocertinib (Exkivity), received accelerated approvalTrusted Source from the Food and Drug Administration (FDA) for the treatment of locally advanced or metastatic NSCLC with EGFR exon 20 insertion mutations. The oral therapy was approved for use in the U.K. in November 2022.

Prof. Siow Ming Lee, professor of medical oncology at University College London, and consultant medical oncologist at University College London Hospitals (UCLH) told Medical News Today:

“NSCLC patients with the uncommon EGFR+ exon 20 insertion mutations have an unmet need. […] Mobocertinib is a first-in-class, oral tyrosine kinase inhibitor (TKI) specifically designed to selectively target the uncommon epidermal growth factor receptor (EGFR) Exon20 insertion mutations.”

Global withdrawal of mobocertinib: What it means

Dr. Gini Harrison, psychologist and research trustee at EGFR+ UK, and EGFR+ survivor, also emphasized how withdrawing mobocertinib from use would severely impact people who rely on it the most.

She explained:

“Mobocertinib is currently the only drug in the U.K. that is licensed for use with this patient group in an NHS [National Health Service] setting. Removing this drug from the market will mean that these patients have no treatment options beyond chemotherapy, which will certainly reduce lifespans and increase mortality rates.”

In early clinical trials, mobocertinib was well tolerated by patients, stopped their cancer from worsening, and increased their survival time, leading to optimism about its potential for people with the exon 20 mutation.

However, in October 2023, the manufacturer, Takeda, voluntarily withdrew MobocertinibTrusted Source from use in the United States.

In phase 3 clinical trials, the drug failed to show a significant effect on progression-free survival (PFS). During the trial, 17% of participants stopped the treatment, half of the patients had to take a break in treatment, and 25% needed the dose reduced because of side effectsTrusted Source, although there were no significant safety concerns.

Takeda said in a press release that it was withdrawing the drug because “the Phase 3 EXCLAIM-2 confirmatory trial, […] did not meet its primary endpoint and thus did not fulfil the confirmatory data requirements of the Accelerated Approval granted by the U.S. FDA nor the conditional marketing approvals granted in other countries.”

However, Prof. Lee explained that the trial had its own shortcomings.

“It is unfortunate that when Takeda designed the first-line trial, the investigator did not include a separate trial arm combining Mobocertinib with chemotherapy,“ he told us.

“Instead, they tested a mobocertinib monotherapy arm against standard chemotherapy, despite knowing that the best ORR [tumor objective response rate] achieved as a second line was approximately 30% in pre-treated NSCLC patients,” Prof. Lee noted.

How will lung cancer patients in the UK be affected?

And if health authorities do withdraw this drug from use in the U.K.? Prof. Lee warned:

“Patients relapsing after first-line platinum chemotherapy will no longer be able to access the drug after the official withdrawal of the conditional marketing authorization for mobocertinib, which will occur in March 2024.”

Dr. Harrison also expressed her frustration to MNT, saying that “[t]he withdrawal of Mobocertinib is purely based on the fact that the drug failed to meet its clinical endpoint in a recent clinical trial.“

”It is not being withdrawn due to safety concerns, and indeed, no new safety concerns have arisen since the drug received its initial licence,” she emphasized.

“Instead,” Dr. Harrison explained, “the drug is being withdrawn because it was licensed on the proviso that a positive result was achieved in an RCT [randomised control trial], showing it to be more efficacious than chemotherapy in a first line setting.”

”The recent RCT (EXCLAIM-2) [trial] showed this was not achieved; however, [the drug] was shown to be as effective as chemotherapy in this setting. Given the effectiveness is on par with a licensed, effective treatment, withdrawing the drug entirely seems both unnecessary and harmful to patients,” she added.

Although mobocertinib will not be available for new patients going forward, Prof. Lee gave some reassurance to those already taking the medication.

“Patients initiated on treatment with mobocertinib before its withdrawal will still be able to access the drug through a compassionate use programme free of charge as long as they are deriving clinical benefit from the medication,” he said.

Call for alternative treatment to be made available in UK

There are alternatives to Mobocertinib in the U.S., but it is the only treatment funded by the National Health Service (NHS) for exon 20 patients in the U.K.

One other drug licensed for use in the U.K. that has shown potential for exon 20 — aminvantamab — is available only privately, so cannot be accessed by those without private insurance or other means of funding, as Dr. Harrison explained.

AmivantamabTrusted Source is an efficacious drug that is used in standard practice for exon 20 patients in Europe and the USA. It is actually approved for private use by the MHRA in the U.K., but is not currently licensed by NICE. While NICE recognises Amivantamab is likely to be efficacious for exon 20, they deem it too expensive to offer it on the NHS.”– Dr. Gini Harrison

She called for the drug to be made available to NHS patients: “[r]ecent randomised controlled trials have shown that amivantamab is likely to be a very efficacious alternative to mobocertinib.”

“Given that this drug is already available privately in the U.K., we suggest that there should be a way to expedite NICE approval processes in situations where the withdrawal of a drug from the market leaves a treatment gap and an unmet need for patients,” she added.

https://www.medicalnewstoday.com/articles/mobocertinib-withdrawal-what-it-means-for-people-with-egfr-lung-cancer#Call-for-alternative-treatment-to-be-made-available-in-UK